Why does trehalose not improve autophagy in the SOD1G93A transgenic mouse model of familial ALS?
Amyotrophic lateral sclerosis is one of the major neurodegenerative diseases, causing an ascending paralysis that usually kills the patient within a few years from disease onset. The motor neurons show aggregates of proteins which in approximately 20 % of cases of the familial form contain mutated S...
| Autor principal: | |
|---|---|
| Formato: | H3 |
| Lenguaje: | Inglés sueco |
| Publicado: |
SLU/Dept. of Biomedical Sciences and Veterinary Public Health (until 231231)
2015
|
| Materias: |
Ejemplares similares: Why does trehalose not improve autophagy in the SOD1G93A transgenic mouse model of familial ALS?
- Not just candy: A herbivore-induced defence-related plant protein in honeydew enhances natural enemy fitness
- Membrane-permeable trehalose 6-phosphate precursor spray increases wheat yields in field trials
- Genetically engineered trehalose accumulation improves cryopreservation tolerance of chrysanthemum (Dendrathema grandiflorum Kitham.) shoot tips
- IFNG-mediated immune responses enhance autophagy against Mycobacterium tuberculosis antigens in patients with active tuberculosis
- Redox regulation of autophagy in Arabidopsis: The different ROS effects
- ViralPlaque: a Fiji macro for automated assessment of viral plaque statistics