| Sumario: | Twenty-two cassava genotypes and eight controls were evaluated in two cropping
seasons for resistance to cassava mosaic disease (CMD) at the International Institute
of Tropical Agriculture (IITA) fields, located at different ecozones of Nigeria. Disease
incidence (DI) and index of symptom severity data were obtained monthly at
each location and genotype. Symptomatic leaves were also collected during evaluation
at each location, and virus was indexed by amplification in polymerase chain
reaction. Significant differences within and across locations were observed in the
reactions of cassava genotypes to CMD. DI across cassava genotypes was
significantly (p = 0.05) highest in the Ibadan (22.6%), followed by Onne (19.3%).
Generally, plants of clones 96/0860, 96/1439, 96/0160, 96/1089A, 96/1632, 96/1613,
96/1708, 96/0191, 96/0249 and 96/1565 had significantly lower values of DI in each
location. African cassava mosaic virus in single infection was the predominant causal
agent of CMD in IITA experimental fields under study.Twenty-two cassava genotypes and eight controls were evaluated in two cropping
seasons for resistance to cassava mosaic disease (CMD) at the International Institute
of Tropical Agriculture (IITA) fields, located at different ecozones of Nigeria. Disease
incidence (DI) and index of symptom severity data were obtained monthly at
each location and genotype. Symptomatic leaves were also collected during evaluation
at each location, and virus was indexed by amplification in polymerase chain
reaction. Significant differences within and across locations were observed in the
reactions of cassava genotypes to CMD. DI across cassava genotypes was
significantly (p = 0.05) highest in the Ibadan (22.6%), followed by Onne (19.3%).
Generally, plants of clones 96/0860, 96/1439, 96/0160, 96/1089A, 96/1632, 96/1613,
96/1708, 96/0191, 96/0249 and 96/1565 had significantly lower values of DI in each
location. African cassava mosaic virus in single infection was the predominant causal
agent of CMD in IITA experimental fields under study.Twenty-two cassava genotypes and eight controls were evaluated in two cropping
seasons for resistance to cassava mosaic disease (CMD) at the International Institute
of Tropical Agriculture (IITA) fields, located at different ecozones of Nigeria. Disease
incidence (DI) and index of symptom severity data were obtained monthly at
each location and genotype. Symptomatic leaves were also collected during evaluation
at each location, and virus was indexed by amplification in polymerase chain
reaction. Significant differences within and across locations were observed in the
reactions of cassava genotypes to CMD. DI across cassava genotypes was
significantly (p = 0.05) highest in the Ibadan (22.6%), followed by Onne (19.3%).
Generally, plants of clones 96/0860, 96/1439, 96/0160, 96/1089A, 96/1632, 96/1613,
96/1708, 96/0191, 96/0249 and 96/1565 had significantly lower values of DI in each
location. African cassava mosaic virus in single infection was the predominant causal
agent of CMD in IITA experimental fields under study.
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