Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function

Theileria parva causes an acute lympho-proliferative disease in cattle, which can result in death of susceptible animals within 2-3 weeks of infection. Analyses of the cellular response in the lymph node draining the site of infection demonstrated an early T cell response, with the appearance of lar...

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Main Authors: Houston, E.F., Taracha, E.L.N., Brackenbury, L., MacHugh, Niall D., McKeever, Declan J., Charleston, B., Morrison, W. Ivan
Format: Journal Article
Language:Inglés
Published: Elsevier 2008
Subjects:
Online Access:https://hdl.handle.net/10568/28788
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author Houston, E.F.
Taracha, E.L.N.
Brackenbury, L.
MacHugh, Niall D.
McKeever, Declan J.
Charleston, B.
Morrison, W. Ivan
author_browse Brackenbury, L.
Charleston, B.
Houston, E.F.
MacHugh, Niall D.
McKeever, Declan J.
Morrison, W. Ivan
Taracha, E.L.N.
author_facet Houston, E.F.
Taracha, E.L.N.
Brackenbury, L.
MacHugh, Niall D.
McKeever, Declan J.
Charleston, B.
Morrison, W. Ivan
author_sort Houston, E.F.
collection Repository of Agricultural Research Outputs (CGSpace)
description Theileria parva causes an acute lympho-proliferative disease in cattle, which can result in death of susceptible animals within 2-3 weeks of infection. Analyses of the cellular response in the lymph node draining the site of infection demonstrated an early T cell response, with the appearance of large numbers of uninfected lymphoblasts between 6 and 9 days p.i., coinciding with initial detection of parasitised cells. There was a marked increase in the representation of CD8+ T cells and the emergence of a sizable sub-population of CD2- CD8+ /ß T cells during this period. Analysis of T cell receptor ß chain variable (TCR BV) gene expression did not reveal any evidence for the involvement of a superantigen in stimulating the response. Responding lymph node cells were found to produce increased quantities of IFN and IL-10, and both the CD2+ CD8+ and CD2- CD8+ populations expressed IFN transcripts. Purified CD2+ CD8+ cells proliferated when stimulated in vitro with autologous parasitised cells or non-specific mitogens, whereas CD2- CD8+ cells were refractory to these stimuli. In contrast to the parasite-specific cytotoxic activity associated with T cell responses in immune cattle, the responses to primary infection exhibited variable levels of non-specific cytotoxic activity. Stimulation of purified CD2+ CD8+ T cells in vitro with autologous parasitised cells also failed to reveal evidence of specific cytotoxic activity. These findings indicate that primary infection with T. parva induces an aberrant T cell response that lacks appropriate effector activity.
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spelling CGSpace287882023-12-08T19:36:04Z Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function Houston, E.F. Taracha, E.L.N. Brackenbury, L. MacHugh, Niall D. McKeever, Declan J. Charleston, B. Morrison, W. Ivan theileria parva cattle infection lymphocytes pathogenesis toxicity cytokines Theileria parva causes an acute lympho-proliferative disease in cattle, which can result in death of susceptible animals within 2-3 weeks of infection. Analyses of the cellular response in the lymph node draining the site of infection demonstrated an early T cell response, with the appearance of large numbers of uninfected lymphoblasts between 6 and 9 days p.i., coinciding with initial detection of parasitised cells. There was a marked increase in the representation of CD8+ T cells and the emergence of a sizable sub-population of CD2- CD8+ /ß T cells during this period. Analysis of T cell receptor ß chain variable (TCR BV) gene expression did not reveal any evidence for the involvement of a superantigen in stimulating the response. Responding lymph node cells were found to produce increased quantities of IFN and IL-10, and both the CD2+ CD8+ and CD2- CD8+ populations expressed IFN transcripts. Purified CD2+ CD8+ cells proliferated when stimulated in vitro with autologous parasitised cells or non-specific mitogens, whereas CD2- CD8+ cells were refractory to these stimuli. In contrast to the parasite-specific cytotoxic activity associated with T cell responses in immune cattle, the responses to primary infection exhibited variable levels of non-specific cytotoxic activity. Stimulation of purified CD2+ CD8+ T cells in vitro with autologous parasitised cells also failed to reveal evidence of specific cytotoxic activity. These findings indicate that primary infection with T. parva induces an aberrant T cell response that lacks appropriate effector activity. 2008-12 2013-05-06T07:01:25Z 2013-05-06T07:01:25Z Journal Article https://hdl.handle.net/10568/28788 en Limited Access Elsevier International Journal for Parasitology;38(14): 1693-1704
spellingShingle theileria parva
cattle
infection
lymphocytes
pathogenesis
toxicity
cytokines
Houston, E.F.
Taracha, E.L.N.
Brackenbury, L.
MacHugh, Niall D.
McKeever, Declan J.
Charleston, B.
Morrison, W. Ivan
Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function
title Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function
title_full Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function
title_fullStr Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function
title_full_unstemmed Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function
title_short Infection of cattle with Theileria parva induces an early CD8 T cell response lacking appropriate effector function
title_sort infection of cattle with theileria parva induces an early cd8 t cell response lacking appropriate effector function
topic theileria parva
cattle
infection
lymphocytes
pathogenesis
toxicity
cytokines
url https://hdl.handle.net/10568/28788
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