Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali

Background The long-lasting insecticidal nets (LLINs) and indoor residual spraying of insecticide (IRS) are major malaria vector control strategies in Mali. The success of control strategies depends on a better understanding of the status of malaria vectors with respect to the insecticides used. In...

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Main Authors: Keïta, M., Kané, F., Thiero, O., Traoré, B., Zeukeng, F., Sodio, A.B., Traoré, S.F., Djouaka, R.F., Doumbia, S., Sogoba, N.
Format: Journal Article
Language:Inglés
Published: Springer 2020
Subjects:
Online Access:https://hdl.handle.net/10568/109660
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author Keïta, M.
Kané, F.
Thiero, O.
Traoré, B.
Zeukeng, F.
Sodio, A.B.
Traoré, S.F.
Djouaka, R.F.
Doumbia, S.
Sogoba, N.
author_browse Djouaka, R.F.
Doumbia, S.
Kané, F.
Keïta, M.
Sodio, A.B.
Sogoba, N.
Thiero, O.
Traoré, B.
Traoré, S.F.
Zeukeng, F.
author_facet Keïta, M.
Kané, F.
Thiero, O.
Traoré, B.
Zeukeng, F.
Sodio, A.B.
Traoré, S.F.
Djouaka, R.F.
Doumbia, S.
Sogoba, N.
author_sort Keïta, M.
collection Repository of Agricultural Research Outputs (CGSpace)
description Background The long-lasting insecticidal nets (LLINs) and indoor residual spraying of insecticide (IRS) are major malaria vector control strategies in Mali. The success of control strategies depends on a better understanding of the status of malaria vectors with respect to the insecticides used. In this study we evaluate the level of resistance of Anopheles gambiae (sensu lato) to bendiocarb and the molecular mechanism that underlies it. Methods Larvae of An. gambiae (s.l.) were collected from breeding habitats encountered in the three study sites and bioassayed with bendiocarb. The ace-1 target site substitution G119S was genotyped using a TaqMan assay. Results The three species of the An. gambiae complex in Mali, i.e. An. arabiensis, An. coluzzii and An. gambiae (s.s.) were found in sympatry in the three surveyed localities with different frequencies. We observed a resistance and suspicious resistance of the three species to bendiocarb with a mortality rate ranging from 37% to 86%. The allelic frequency of the G119S mutation was higher in An. gambiae (s.s.) compared to the other two species; 42.86%, 25.61% and 16.67% respectively in Dangassa, Koula, and Karadié. The allelic frequency of G119S in An. coluzzii ranged from 4.5% to 8.33% and from 1.43% to 21.15% for An. arabiensis. After exposure to bendiocarb, the G119S mutation was found only in survivors. The survival of Anopheles gambiae (s.l) populations from the three surveyed localities was associated with the presence of the mutation. Conclusions The study highlights the implication of G119S mutation in bendiocarb resistance in An. gambiae (s.s.), An. arabiensis and An. coluzzii populations from the three surveyed localities.
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spelling CGSpace1096602025-11-11T10:08:11Z Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali Keïta, M. Kané, F. Thiero, O. Traoré, B. Zeukeng, F. Sodio, A.B. Traoré, S.F. Djouaka, R.F. Doumbia, S. Sogoba, N. malaria mali anopheles gambiae vectors defence mechanisms infectious diseases parasitology Background The long-lasting insecticidal nets (LLINs) and indoor residual spraying of insecticide (IRS) are major malaria vector control strategies in Mali. The success of control strategies depends on a better understanding of the status of malaria vectors with respect to the insecticides used. In this study we evaluate the level of resistance of Anopheles gambiae (sensu lato) to bendiocarb and the molecular mechanism that underlies it. Methods Larvae of An. gambiae (s.l.) were collected from breeding habitats encountered in the three study sites and bioassayed with bendiocarb. The ace-1 target site substitution G119S was genotyped using a TaqMan assay. Results The three species of the An. gambiae complex in Mali, i.e. An. arabiensis, An. coluzzii and An. gambiae (s.s.) were found in sympatry in the three surveyed localities with different frequencies. We observed a resistance and suspicious resistance of the three species to bendiocarb with a mortality rate ranging from 37% to 86%. The allelic frequency of the G119S mutation was higher in An. gambiae (s.s.) compared to the other two species; 42.86%, 25.61% and 16.67% respectively in Dangassa, Koula, and Karadié. The allelic frequency of G119S in An. coluzzii ranged from 4.5% to 8.33% and from 1.43% to 21.15% for An. arabiensis. After exposure to bendiocarb, the G119S mutation was found only in survivors. The survival of Anopheles gambiae (s.l) populations from the three surveyed localities was associated with the presence of the mutation. Conclusions The study highlights the implication of G119S mutation in bendiocarb resistance in An. gambiae (s.s.), An. arabiensis and An. coluzzii populations from the three surveyed localities. 2020-12 2020-09-25T11:29:16Z 2020-09-25T11:29:16Z Journal Article https://hdl.handle.net/10568/109660 en Open Access application/pdf Springer Keïta, M., Kané, F., Thiero, O., Traoré, B., Zeukeng, F., Sodio, A.B., ... & Sogoba, N. (2020). Acetylcholinesterase (ace-1 R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali. Parasites & Vectors, 13(1), 1-9.
spellingShingle malaria
mali
anopheles gambiae
vectors
defence mechanisms
infectious diseases
parasitology
Keïta, M.
Kané, F.
Thiero, O.
Traoré, B.
Zeukeng, F.
Sodio, A.B.
Traoré, S.F.
Djouaka, R.F.
Doumbia, S.
Sogoba, N.
Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali
title Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali
title_full Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali
title_fullStr Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali
title_full_unstemmed Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali
title_short Acetylcholinesterase (ace‑1R) target site mutation G119S and resistance to carbamates in Anopheles gambiae (sensu lato) populations from Mali
title_sort acetylcholinesterase ace 1r target site mutation g119s and resistance to carbamates in anopheles gambiae sensu lato populations from mali
topic malaria
mali
anopheles gambiae
vectors
defence mechanisms
infectious diseases
parasitology
url https://hdl.handle.net/10568/109660
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