Showing 1 - 20 results of 111 for search '"Tumor Necrosis Factor-alpha/metabolism"', query time: 1.43s Refine Results
1

Contributors: Physiotherapy, Human Physiology and Anatomy, Brussels Heritage Lab, Pain in Motion, Faculty of Physical Education and Physical Therapy, Movement and Sport Sciences, Physical Medicine and Rehabilitation, Internal Medicine, Clinical sciences

File Description: Electronic

2
3
4
5
6

Source: Cellular and molecular life sciences, vol. 78, no. 1, pp. 227-247
Cellular and Molecular Life Sciences

File Description: application/pdf

7
8

Source: EBioMedicine, Vol 24, Iss C, Pp 195-204 (2017)
EBioMedicine
EBioMedicine, vol. 24, pp. 195-204

File Description: application/pdf

9
10

Contributors: University of Zurich, Conrad, Curdin

Source: Nature Communications, Vol 9, Iss 1, Pp 1-11 (2018)
Nature communications, vol. 9, no. 1, pp. 25
Nature Communications

Subject Terms: General Physics and Astronomy, General Biochemistry, Genetics and Molecular Biology, General Chemistry, Immunology, Adalimumab, medicine.drug, medicine, Interferon, business.industry, business, Infliximab, Interferon type I, Autoimmunity, medicine.disease_cause, Type I interferon production, Tumor necrosis factor alpha, Psoriasis, medicine.disease, lcsh:Science, lcsh:Q, Science, Adalimumab/adverse effects, Adalimumab/immunology, Adalimumab/therapeutic use, Adolescent, Adult, Aged, Animals, Antibodies, Monoclonal/adverse effects, Antibodies, Monoclonal/immunology, Antibodies, Monoclonal/therapeutic use, Autoimmunity/drug effects, Autoimmunity/immunology, Cells, Cultured, Crohn Disease/drug therapy, Crohn Disease/immunology, Crohn Disease/metabolism, Cytokines/genetics, Cytokines/immunology, Cytokines/metabolism, Dendritic Cells/drug effects, Dendritic Cells/immunology, Dendritic Cells/metabolism, Female, Humans, Infliximab/adverse effects, Infliximab/immunology, Infliximab/therapeutic use, Interferon Type I/genetics, Interferon Type I/immunology, Interferon Type I/metabolism, Male, Mice, Inbred BALB C, Middle Aged, Psoriasis/chemically induced, Psoriasis/immunology, T-Lymphocytes/drug effects, T-Lymphocytes/immunology, T-Lymphocytes/metabolism, Tumor Necrosis Factor-alpha/antagonists & inhibitors, Tumor Necrosis Factor-alpha/immunology, Tumor Necrosis Factor-alpha/metabolism, Young Adult, 610 Medicine & health, 10177 Dermatology Clinic, 1600 General Chemistry, 1300 General Biochemistry, Genetics and Molecular Biology, 3100 General Physics and Astronomy, Article

File Description: application/pdf; 2018_LF_TNF_blockade_induces_a_dysregulated_type_I_interferon_response_without_autoimmunity_in_paradoxical_psoriasis.pdf - application/pdf

11

Contributors: Centre d’Infection et d’Immunité de Lille - INSERM U 1019 - UMR 9017 - UMR 8204 (CIIL), Centre National de la Recherche Scientifique (CNRS)-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)-Université de Lille-Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), Faculté des Sciences, Université Libanaise, Universiteit Gent = Ghent University [Belgium] (UGENT), VIB-UGent Center for Inflammation Research [Gand, Belgique] (IRC), VIB [Belgium], Groningen Biomolecular Sciences and Biotechnology Institute (GBB), University of Groningen [Groningen], Hannover Medical School [Hannover] (MHH), Immunologie et Neurogénétique Expérimentales et Moléculaires (INEM), Université d'Orléans (UO)-Centre National de la Recherche Scientifique (CNRS), University of Cape Town, Institute of Infection and Global Health [University of Liverpool, UK], University of Liverpool, C.P., J.-C.S., and C.F. were supported by INSERM. B.R. and F.T. were supported by CNRS. Work in M.L.’s laboratory is supported by VIB, Ghent University (BOF 01N02313, BOF 01J11113, BOF14/GOA/013), the Fund for Scientific Research-Flanders (grants G030212N and G011315N), and the European Research Council (grant 281600). M.H. was the recipient of a doctoral fellowship from the AZM foundation. E.C.P. was supported by a post-doctoral fellowship from the French Institute of cancer (INCa)., Dr Isabelle Wolowczuk (CIIL, Institut Pasteur, Lille) is acknowledged for critical reading of this manuscript. We thank Nathalie Messéant and Bruno Couvreur for mouse husbanding. We also thank Aurélie Maillard, Pauline Chenuet, and the BICeL flow cytometry core facility for technical assistance., Centre d’Etude des Pathologies Respiratoires (CEPR), UMR 1100 (CEPR), Université de Tours (UT)-Institut National de la Santé et de la Recherche Médicale (INSERM), Molecular Genetics, Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)-Centre National de la Recherche Scientifique (CNRS), Université de Tours-Institut National de la Santé et de la Recherche Médicale (INSERM), Centre National de la Recherche Scientifique (CNRS)-Université d'Orléans (UO)

Source: Mucosal Immunology
Mucosal Immunology, Nature Pub. Group, 2017, 10 (4), pp.1056-1068. ⟨10.1038/mi.2016.113⟩
Mucosal immunology, 10(4), 1056-1068. Nature Publishing Group
Mucosal Immunology, 2017, 10 (4), pp.1056-1068. ⟨10.1038/mi.2016.113⟩

File Description: application/pdf

12

Contributors: University of Zurich, Volterra, Andrea

Source: Cell
Cell, vol. 163, no. 7, pp. 1730-1741

File Description: application/pdf; 2015_Cell20151631730.pdf - application/pdf; Habbas_etal._2015.pdf - application/pdf

13

Contributors: Bedrijfsbureau CD, RS: CARIM - R3 - Vascular biology, Castoldi, G, di Gioia, C, Bombardi, C, Maestroni, S, Carletti, R, Muscha Steckelings, U, Dahlöf, B, Unger, T, Zerbini, G, Stella, A

Source: Castoldi, G, di Gioia, C R, Bombardi, C, Maestroni, S, Carletti, R, Steckelings, U M, Dahlöf, B, Unger, T, Zerbini, G & Stella, A 2014, ' Prevention of diabetic nephropathy by compound 21, selective agonist of angiotensin type 2 receptors, in Zucker diabetic fatty rats ', American Journal of Physiology: Renal Physiology, vol. 307, no. 10, pp. F1123-F1131 . https://doi.org/10.1152/ajprenal.00247.2014
American Journal of Physiology-Renal Physiology, 307(10), F1123-F1131. American Physiological Society

14

Source: Lildal, S K, Nørregaard, R, Andreassen, K H, Christiansen, F E, Jung, H U, Pedersen, M R V & Osther, P J S 2017, ' Ureteral Access Sheath Influence on the Ureteral Wall Evaluated by Cyclooxygenase-2 and Tumor Necrosis Factor-α in a Porcine Model ', Journal of Endourology, vol. 31, no. 3, pp. 307-313 . https://doi.org/10.1089/end.2016.0773
Journal of Endourology
Lildal, S K, Norregaard, R, Andreassen, K H, Christiansen, F E, Jung, H, Pedersen, M R & Osther, P J S 2017, ' Ureteral Access Sheath Influence on the Ureteral Wall Evaluated by Cyclooxygenase-2 and Tumor Necrosis Factor-in a Porcine Model ', Journal of Endourology, vol. 31, no. 3, pp. 307-313 . https://doi.org/10.1089/end.2016.0773

File Description: application/pdf

15

Source: International Journal of Molecular Sciences; Volume 18; Issue 2; Pages: 326
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 18, Iss 2, p 326 (2017)

File Description: application/pdf

16
17

Source: Clausen, B H, Degn, M, Sivasaravanaparan, M, Fogtmann, T, Andersen, M G, Trojanowsky, M D, Gao, H, Hvidsten, S, Baun, C, Deierborg, T, Finsen, B, Kristensen, B W, Bak, S T, Meyer, M, Lee, J, Nedospasov, S A, Brambilla, R & Lambertsen, K L 2016, ' Conditional ablation of myeloid TNF increases lesion volume after experimental stroke in mice, possibly via altered ERK1/2 signaling ', Scientific Reports, vol. 6, 29291 . https://doi.org/10.1038/srep29291
Scientific Reports

File Description: application/pdf

18

Source: Madsen, P M, Motti, D, Karmally, S, Szymkoski, D E, Lambertsen, K L, Bethea, J R & Brambilla, R 2016, ' Oligodendroglial TNFR2 mediates membrane TNF-dependent repair in experimental autoimmune encephalomyelitis by promoting oligodendrocyte differentiation and remyelination ', The Journal of Neuroscience, vol. 36, no. 18, pp. 5128-5143 . https://doi.org/10.1523/JNEUROSCI.0211-16.2016

File Description: application/pdf

19

Contributors: Lille Inflammation Research International Center - U 995 (LIRIC), Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), Centre de Recherche Jean-Pierre AUBERT Neurosciences et Cancer - U1172 Inserm - U837 (JPArc), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Lille Nord de France (COMUE)-Université de Lille, Mécanismes de la Tumorigénèse et Thérapies Ciblées - UMR 8161 (M3T), Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre National de la Recherche Scientifique (CNRS), Centre Hospitalier Universitaire de Lille (CHU de Lille), Chirurgie digestive et transplantation [Lille], Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)-Espace éthique hospitalier et universitaire de Lille - EEHU, Inserm, Université de Lille, CHU Lille, CNRS, Lille Inflammation Research International Center (LIRIC) - U995, Centre de Recherche Jean-Pierre AUBERT Neurosciences et Cancer (JPArc) - U1172, Mécanismes de la Tumorigénèse et Thérapies Ciblées (M3T) - UMR 8161, Lille Inflammation Research International Center [LIRIC], Centre de Recherche Jean-Pierre AUBERT Neurosciences et Cancer [JPArc - U837 Inserm], Centre de Recherche Jean-Pierre AUBERT Neurosciences et Cancer - U837 (JPArc), Université Lille Nord de France (COMUE)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille, Lille Inflammation Research International Center - U 995 [LIRIC], Lille Neurosciences & Cognition (LilNCog) - U 1172, Mécanismes de la Tumorigénèse et Thérapies Ciblées - UMR 8161 [M3T]

Source: Gut
Gut, BMJ Publishing Group, 2015, Gut, 64, pp.1949-1960. ⟨10.1136/gutjnl-2014-308410⟩
Gut, 2015, Gut, 64, pp.1949-1960. ⟨10.1136/gutjnl-2014-308410⟩

File Description: application/octet-stream

20