Showing 1 - 20 results of 84 for search '"Anti-Inflammatory Agents pharmacology"', query time: 1.43s Refine Results
1

Contributors: Supporting clinical sciences, Medical Imaging, Department of Bio-engineering Sciences, Cellular and Molecular Immunology

Source: Frontiers in Immunology, Vol 12 (2021)
Frontiers in Immunology

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Contributors: Institut de Recherche en Santé Digestive (IRSD ), Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Ecole Nationale Vétérinaire de Toulouse (ENVT), Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut National de Recherche pour l’Agriculture, l’Alimentation et l’Environnement (INRAE), Institut Toulousain des Maladies Infectieuses et Inflammatoires (Infinity), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), association Francois Aupetit, AFA2019/Association Francois Aupetit, SEGUIN, Nathalie

Source: International Journal of Molecular Sciences
International Journal of Molecular Sciences, MDPI, 2021, 22 (14), 14 p. ⟨10.3390/ijms22147387⟩
International Journal of Molecular Sciences, 2021, 22 (14), pp.7387. ⟨10.3390/ijms22147387⟩
International Journal of Molecular Sciences, Vol 22, Iss 7387, p 7387 (2021)
Volume 22
Issue 14

Subject Terms: CD4-Positive T-Lymphocytes, MESH: Colitis / chemically induced, [SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO], colitis, Narcotic Antagonists, MESH: Animals, Deraison, MESH: Anti-Inflammatory Agents, Non-Steroidal / pharmacology, Anti-Inflammatory Agents, MESH: Epithelial Cells / drug effects, [SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry, Molecular Biology/Molecular biology, Severity of Illness Index, M, Blanpied, Mice, MESH: Inflammation / metabolism, A, Biology (General), Intestinal Mucosa, MESH: Apoptosis / genetics, Mice, Knockout, [SDV.MHEP] Life Sciences [q-bio]/Human health and pathology, Naloxone, scolitis, Anti-Inflammatory Agents, Non-Steroidal, Saoudi, MESH: CD4-Positive T-Lymphocytes / drug effects, apoptosis, MESH: Apoptosis / drug effects, Cenac, Interleukin-10, Chemistry, Mas-Orea, Petitfils, Sebert, Cytokines, G. Peripheral Opioid Receptor Blockade Enhances opioids, QH301-705.5, [SDV.BC]Life Sciences [q-bio]/Cellular Biology, Article, Permeability, MESH: Colitis / genetics, C, MESH: Anti-Inflammatory Agents / pharmacology, [SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO], Animals, Dietrich, X, QD1-999, [SDV.BC] Life Sciences [q-bio]/Cellular Biology, Benamar, Barreau, Inflammation, intestinal permeability, piroxicam, MESH: Cytokines / metabolism, MESH: Inflammation / genetics, opioids, Epithelial Cells, [SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Molecular biology, N, MESH: Cytokines / genetics, Mice, Inbred C57BL, Quaternary Ammonium Compounds, IL-10-deficient mice, F, Receptors, Opioid, opioid, MESH: Colitis / metabolism, MESH: Colitis / pathology, [SDV.MHEP]Life Sciences [q-bio]/Human health and pathology

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Source: Endocrine, Metabolic & Immune Disorders - Drug Targets. 19:1141-1147

Subject Terms: Lipopolysaccharides, 0301 basic medicine, Lipopolysaccharide, cell infiltration, Endocrinology, Diabetes and Metabolism, pancreatitis, Anti-Inflammatory Agents, Pregabalin, Nitric Oxide Synthase Type II, Wistar rat, immune response, granulocyte colony stimulating factor, sepsis, Pathogenesis, chemistry.chemical_compound, 0302 clinical medicine, biochemical analysis, caspase 3, Nos2 protein, rat, Granulocyte Colony-Stimulating Factor, Pathology, Immunology and Allergy, rat, animal, pancreas, glucose, biology, Caspase 3, lipopolysaccharide, drug effect, single drug dose, Immunohistochemistry, Nitric oxide synthase, female, medicine.anatomical_structure, 030220 oncology & carcinogenesis, histopathology, Female, lipids (amino acids, peptides, and proteins), Inflammation Mediators, Pancreas, antiinflammatory agent, signal transduction, Signal Transduction, medicine.drug, amylase, medicine.medical_specialty, autacoid, cell protection, Casp3 protein, rat, animal experiment, interleukin 6, Granulocyte, Il6 protein, rat, Article, leucocyte infiltration, animal tissue, Sepsis, 03 medical and health sciences, Animals, Anti-Inflammatory Agents/*pharmacology, Caspase 3/metabolism, Cytoprotection, Disease Models, Animal, Granulocyte Colony-Stimulating Factor/metabolism, Inflammation Mediators/metabolism, Interleukin-6/metabolism, Nitric Oxide Synthase Type II/metabolism, Pancreas/*drug effects/metabolism/pathology, Pancreatitis/blood/chemically induced/pathology/*prevention & control, Pregabalin/*pharmacology, Rats, Wistar, Serum Amyloid A Protein/metabolism, Signal Transdu, blood, Internal medicine, medicine, Endocrine system, controlled study, neutrophil chemotaxis, protein expression, Serum Amyloid A Protein, nonhuman, Interleukin-6, business.industry, animal model, disease model, aging, inducible nitric oxide synthase, serum amyloid A, triacylglycerol lipase, medicine.disease, 030104 developmental biology, Endocrinology, Pancreatitis, chemistry, biology.protein, business, metabolism

4

Source: Annals of Cardiac Anaesthesia
Annals of Cardiac Anaesthesia, Vol 22, Iss 4, Pp 372-378 (2019)

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5

Source: Andersen, T, Østgård, R D, Aspari, M P, Kragstrup, T W, Glerup, H, Jurik, A G, Johansen, C, Hvid, M & Deleuran, B W 2019, ' Anti-tumor necrosis factor treatment increases both the Th17 and Th22 T helper subsets in spondyloarthritis ', APMIS, vol. 127, no. 12, pp. 789-796 . https://doi.org/10.1111/apm.12994

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7

Contributors: Faculty of Medicine and Pharmacy, Pharmaceutical and Pharmacological Sciences, Experimental in vitro toxicology and dermato-cosmetology, Connexin Signalling Research Group, Liver Connexin and Pannexin Research Group

Source: Repositório Institucional da USP (Biblioteca Digital da Produção Intelectual)
Universidade de São Paulo (USP)
instacron:USP

8

Contributors: Institut de Génétique Moléculaire de Montpellier (IGMM), Centre National de la Recherche Scientifique (CNRS)-Université de Montpellier (UM), Institut de Recherche en Cancérologie de Montpellier (IRCM - U1194 Inserm - UM), CRLCC Val d'Aurelle - Paul Lamarque-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Montpellier (UM), Université de Montpellier (UM)-Centre National de la Recherche Scientifique (CNRS), Salvy-Córdoba, Nathalie

Source: Scientific Reports, Vol 7, Iss 1, Pp 1-11 (2017)
Scientific Reports
Scientific Reports, Nature Publishing Group, 2017, 7 (1), ⟨10.1038/s41598-017-04071-3⟩
Scientific Reports, 2017, 7 (1), ⟨10.1038/s41598-017-04071-3⟩

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9

Contributors: Laboratory of Plant Biotechnology [Sfax], Faculté des Sciences de Sfax, Université de Sfax - University of Sfax-Université de Sfax - University of Sfax, Laboratoire des Venins et Biomolécules Thérapeutiques - Laboratory of Venoms and Therapeutic Biomolecules (LR11IPT08), Institut Pasteur de Tunis, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), Université de Tunis El Manar (UTM), This research was supported by the Ministry of Higher Education and Scientific Research in Tunisia., The major part of this work was accomplished at the Faculty of Sciences of Sfax and at the Institute Pasteur of Tunis. The authors gratefully acknowledge the support of Mr. El Abed Mokhles for his assistance in the project.

Source: BioMed Research International
BioMed Research International, Hindawi Publishing Corporation, 2018, 2018, pp.1542602. ⟨10.1155/2018/1542602⟩
BioMed Research International, Vol 2018 (2018)

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11

Contributors: Swiss IBD Cohort Study Group, University of Zurich, Zeitz, Jonas

Source: PloS one, vol. 14, no. 4, pp. e0215738
PLoS ONE
PLoS ONE, Vol 14, Iss 4, p e0215738 (2019)

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Contributors: Université Abderrahmane Mira [Béjaïa], Faculté de médecine de Sousse [Ibn EL Jazzar], Laboratoire Bio-PeroxIL. Biochimie du Peroxysome, Inflammation et Métabolisme Lipidique ( Bio-PeroxIL ), Université de Bourgogne ( UB ), Nutrition, Aliments Fonctionnels et Santé Vasculaire, Université de Monastir ( UM ), Institut Pasteur de Tunis, Université Mohamed Akli Ouelhadj Bouira (UMAOB), Laboratoire Bio-PeroxIL. Biochimie du Peroxysome, Inflammation et Métabolisme Lipidique (Bio-PeroxIL), Université de Bourgogne (UB), Université de Monastir - University of Monastir (UM), Laboratoire des Venins et Biomolécules Thérapeutiques - Laboratory of Venoms and Therapeutic Biomolecules (LR11IPT08), Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), Université Mohamed Akli Ouelhadj de Bouira (UMAOB), This work was supported by the University of Bourgogne and INSERM., We thank Prof. Norbert Latruffe (Univ. of Bourgogne, EA7270) for his helpful comments on the manuscript. The authors thank Mr Philip Bastable (Pôle Recherche – Délégation à la Recherche Clinique et Innovation – CHU Dijon, France) for English corrections.

Source: Pharmaceutical Biology
Pharmaceutical Biology, Taylor & Francis, 2017, 55 (1), pp.286-293. 〈10.1080/13880209.2016.1235208〉
Pharmaceutical Biology, Taylor & Francis, 2017, 55 (1), pp.286-293. ⟨10.1080/13880209.2016.1235208⟩
Pharmaceutical Biology, Vol 55, Iss 1, Pp 286-293 (2017)
Scopus-Elsevier

Subject Terms: 0301 basic medicine, MESH: Monocytes/pathology, Time Factors, Anti-Inflammatory Agents, Pharmaceutical Science, MESH: Mentha/chemistry, Apoptosis, MESH: Plants, Medicinal, Monocytes, MESH: Dose-Response Relationship, Drug, Mice, Drug Discovery, Cytotoxic T cell, MESH: RAW 264.7 Cells, mentha rotundifolia, MESH: Animals, Cytotoxicity, MESH: Monocytes/metabolism, MESH: Inhibitory Concentration 50, Membrane Potential, Mitochondrial, U937 cell, Caspase 3, Biological activity, General Medicine, U937 Cells, MESH: Membrane Potential, Mitochondrial/drug effects, mitochondria, MESH: Phytotherapy, MESH: Plant Extracts/isolation & purification, Molecular Medicine, MESH: Autophagy/drug effects, MESH: Plant Extracts/pharmacology, Microtubule-Associated Proteins, MESH: Apoptosis/drug effects, Mentha, mentha pulegium, Stereochemistry, Cell Survival, Context (language use), Biology, MESH: U937 Cells, Mentha spicata, MESH: Monocytes/drug effects, 03 medical and health sciences, Inhibitory Concentration 50, [ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathology, Autophagy, Animals, Humans, MTT assay, [SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology, MESH: Macrophages/metabolism, MESH: Macrophages/drug effects, MESH: Mice, Pharmacology, Plants, Medicinal, MESH: Humans, Dose-Response Relationship, Drug, Plant Extracts, Macrophages, lcsh:RM1-950, MESH: Time Factors, MESH: Anti-Inflammatory Agents/isolation & purification, Molecular biology, [SDV.BV.PEP]Life Sciences [q-bio]/Vegetal Biology/Phytopathology and phytopharmacy, 030104 developmental biology, lcsh:Therapeutics. Pharmacology, RAW 264.7 Cells, Complementary and alternative medicine, MESH: Macrophages/pathology, MESH: Microtubule-Associated Proteins/metabolism, MESH: Anti-Inflammatory Agents/pharmacology, Mentha pulegium, MESH: Caspase 3/metabolism, MESH: Cell Survival/drug effects, Phytotherapy

14

Contributors: College of Dentistry, Taibah University, Laboratory for Research on Biologically Compatible Compounds, Faculty of Dental Medicine of Monastir, Laboratoire de Transmission, Contrôle et Immunobiologie des Infections ( LR11IPT02 ), Institut Pasteur de Tunis-Réseau International des Instituts Pasteur ( RIIP ), Université Tunis El Manar ( UTM ), Faculté de Médecine Dentaire de Monastir [Tunisie] (FMDM), Laboratoire de Transmission, Contrôle et Immunobiologie des Infections - Laboratory of Transmission, Control and Immunobiology of Infection (LR11IPT02), Institut Pasteur de Tunis, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), Université de Tunis El Manar (UTM), Université de Jendouba (UJ)

Source: Biomedicine and Pharmacotherapy
Biomedicine and Pharmacotherapy, Elsevier Masson, 2016, 84, pp.1986-1991. 〈10.1016/j.biopha.2016.11.010 〉
Biomedicine and Pharmacotherapy, Elsevier Masson, 2016, 84, pp.1986-1991. ⟨10.1016/j.biopha.2016.11.010⟩

Subject Terms: Lipopolysaccharides, 0301 basic medicine, Lipopolysaccharide, MESH: Cell Survival/physiology, MESH: Lipopolysaccharides/toxicity, [SDV]Life Sciences [q-bio], Anti-Inflammatory Agents, Drug Evaluation, Preclinical, Nitric Oxide Synthase Type II, Venom, MESH: Cell Proliferation/drug effects, MESH: Leukocytes, Mononuclear/drug effects, MESH: Dose-Response Relationship, Drug, Mice, chemistry.chemical_compound, 0302 clinical medicine, MESH: Animals, MESH: Interferon-gamma/toxicity, MESH: Cnidarian Venoms/isolation & purification, Cell proliferation, ComputingMilieux_MISCELLANEOUS, MTT assay, biology, MESH: Nitric Oxide Synthase Type II/metabolism, General Medicine, [SDV.SP]Life Sciences [q-bio]/Pharmaceutical sciences, 3. Good health, MESH: Drug Evaluation, Preclinical/methods, 030220 oncology & carcinogenesis, MESH: Leukocytes, Mononuclear/metabolism, Cell Survival, Peripheral blood mononuclear cell, MESH: Cnidarian Venoms/pharmacology, Cell Line, Nitric oxide, Microbiology, Interferon-gamma, 03 medical and health sciences, Anti-inflammatory activity, Cnidarian Venoms, Animals, Humans, MESH: Macrophages/metabolism, MESH: Nitric Oxide Synthase Type II/antagonists & inhibitors, MESH: Macrophages/drug effects, MESH: Mice, Pelagia noctiluca, Pharmacology, MESH: K562 Cells, MESH: Humans, Dose-Response Relationship, Drug, [ SDV ] Life Sciences [q-bio], Cell growth, Macrophages, MESH: Cell Proliferation/physiology, biology.organism_classification, MESH: Anti-Inflammatory Agents/isolation & purification, Molecular biology, MESH: Cell Line, 030104 developmental biology, chemistry, Cell culture, Leukocytes, Mononuclear, MESH: Anti-Inflammatory Agents/pharmacology, K562 Cells, MESH: Cell Survival/drug effects, K562 cells

15

Source: Thrombosis and Haemostasis; Vol 111
Thrombosis and Haemostasis, Vol. 111, No 2 (2014) pp. 308-322
Thrombosis and Haemostasis
Thrombosis and haemostasis

Subject Terms: Carotid Artery Diseases, 0301 basic medicine, Piperidines/pharmacology, Time Factors, Chemokine CXCL1, Transcription Factor RelA/metabolism, Anti-Inflammatory Agents, Nicotinamide phosphoribosyltransferase, Pharmacology, Mice, chemistry.chemical_compound, 0302 clinical medicine, Piperidines, Signal Transduction/drug effects, Neutrophil Infiltration/drug effects, Anti-Inflammatory Agents/pharmacology, Enzyme Inhibitors, Nicotinamide Phosphoribosyltransferase, Apolipoproteins E/deficiency/genetics, Cells, Cultured, ddc:616, Enzyme Inhibitors/pharmacology, Mice, Knockout, Cytokines/antagonists & inhibitors/metabolism, Hematology, Plaque, Atherosclerotic, 3. Good health, CXCL1, Carotid Arteries, Matrix Metalloproteinase 9, Neutrophil Infiltration, Carotid Arteries/drug effects/enzymology/immunology/pathology, Cytokines, Collagen, Matrix Metalloproteinase 9/metabolism, medicine.symptom, Infiltration (medical), Atherosclerosis/drug therapy/enzymology/genetics/immunology/pathology, Signal Transduction, Nicotinamide Phosphoribosyltransferase/antagonists & inhibitors/metabolism, Inflammation, Acrylamides/pharmacology, Diet, High-Fat, Chemokine CXCL1/metabolism, 03 medical and health sciences, Apolipoproteins E, In vivo, Collagen/metabolism, Human Umbilical Vein Endothelial Cells, medicine, Animals, Humans, ddc:576, Acrylamides, business.industry, Transcription Factor RelA, Carotid Artery Diseases/drug therapy/enzymology/genetics/immunology/pathology, Human Umbilical Vein Endothelial Cells/drug effects/enzymology/immunology, medicine.disease, In vitro, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, chemistry, inflammation, Ischaemic myocardium, Immunology, atherosclerosis, business, Neutrophil recruitment, Carotid artery, 030217 neurology & neurosurgery

16

Contributors: Hémostase, Inflammation, Thrombose (HITH - U1176 Inserm - CHU Bicêtre), Université Paris-Sud - Paris 11 (UP11)-AP-HP Hôpital Bicêtre (Le Kremlin-Bicêtre)-Institut National de la Santé et de la Recherche Médicale (INSERM), Vascular research center of Marseille (VRCM), Institut National de la Santé et de la Recherche Médicale (INSERM)-Aix Marseille Université (AMU), Service d'hématologie, immunologie biologiques et cytogénétique, Université Paris-Sud - Paris 11 (UP11)-Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Hôpital Bicêtre, Institut de Recherches Internationales Servier [Suresnes] (IRIS), The study was financially supported by a PhD-research grant from Institut Servier & Association Nationale de la Recherche Technique (Contrat CIFRE) to JPe, and an Inserm-Poste d’Accueil-grant (# ASC13101LSA) to MP., Université Paris-Sud - Paris 11 (UP11)-Institut National de la Santé et de la Recherche Médicale (INSERM)-AP-HP Hôpital Bicêtre (Le Kremlin-Bicêtre), Aix Marseille Université (AMU)-Institut National de la Santé et de la Recherche Médicale (INSERM)

Source: Scientific Reports
Scientific Reports, Nature Publishing Group, 2016, 6 (1), pp.37953. ⟨10.1038/srep37953⟩
Scientific Reports, 2016, 6 (1), pp.37953. ⟨10.1038/srep37953⟩

Subject Terms: MESH: Anti-Inflammatory Agents/pharmacology, E-SELECTIN LIGAND-1, [SDV]Life Sciences [q-bio], Anti-Inflammatory Agents, MESH: Membrane Glycoproteins/genetics, MESH: Antigens, Differentiation, Myelomonocytic/metabolism, MESH: Lectins/metabolism, MESH: Antigens, CD/pharmacology, MESH: Leukocytes, Mononuclear/drug effects, Lectins, MESH: Animals, MYELOID CELLS, MESH: E-Selectin/metabolism, NEUTROPHILS, IN-VIVO, MESH: Inflammation/drug therapy, Membrane Glycoproteins, MESH: Inflammation/chemically induced, MESH: Inflammation/pathology, MESH: Membrane Glycoproteins/metabolism, MESH: Leukocyte Rolling/physiology, P-Selectin, Female, E-Selectin, MESH: Leukocytes, Mononuclear/metabolism, CD33-RELATED SIGLECS, VON-WILLEBRAND-FACTOR, MESH: Lectins/pharmacology, Antigens, Differentiation, Myelomonocytic, MESH: Antigens, Differentiation, Myelomonocytic/pharmacology, MESH: Tumor Necrosis Factor-alpha/toxicity, GLYCOPROTEIN LIGAND, MESH: Antigens, CD/metabolism, Article, MESH: Mice, Inbred C57BL, Antigens, CD, Animals, Humans, Leukocyte Rolling, Protein Interaction Domains and Motifs, MESH: Antigens, Differentiation, Myelomonocytic/genetics, SIALIC-ACID, Inflammation, MESH: Protein Interaction Domains and Motifs, MESH: Humans, Tumor Necrosis Factor-alpha, MESH: Lectins/genetics, MESH: Solubility, Mice, Inbred C57BL, Disease Models, Animal, Solubility, Leukocytes, Mononuclear, MESH: Antigens, CD/genetics, IMMUNE-SYSTEM, MESH: Disease Models, Animal, MESH: Female, MESH: P-Selectin/metabolism

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Source: Arteriosclerosis thrombosis and vascular biology
Arteriosclerosis, Thrombosis, and Vascular Biology, Vol. 33, No 9 (2013) pp. 2187-92
ATVB

Subject Terms: Male, Time Factors, Biological Markers/metabolism, Platelet Adhesiveness/drug effects, Vascular Cell Adhesion Molecule-1/metabolism, Anti-Inflammatory Agents, Contrast Media, 030204 cardiovascular system & hematology, ddc:616.07, medicine.disease_cause, Antioxidants, chemistry.chemical_compound, Mice, 0302 clinical medicine, Superoxides, Macrophages/drug effects/metabolism, Anti-Inflammatory Agents/pharmacology, Enzyme Inhibitors, Enzyme Inhibitors/pharmacology, Mice, Knockout, 0303 health sciences, Microbubbles, medicine.diagnostic_test, Superoxide, Cell adhesion molecule, 3. Good health, Molecular Imaging, Phenotype, Biochemistry, Platelet Glycoprotein GPIb-IX Complex, cardiovascular system, Aortic Diseases/drug therapy/genetics/metabolism/pathology/ultrasonography, Molecular Imaging/methods, Superoxides/metabolism, Cardiology and Cardiovascular Medicine, Nicotinamide adenine dinucleotide phosphate, APOBEC-1 Deaminase, Contrast Media/diagnostic use, Blotting, Western, Aortic Diseases, Oxidative Stress/drug effects, Vascular Cell Adhesion Molecule-1, Biology, Article, Endothelial activation, 03 medical and health sciences, Cytidine Deaminase/deficiency/genetics, Platelet Adhesiveness, Western blot, medicine.artery, Cytidine Deaminase, medicine, Animals, Ultrasonography, Interventional, 030304 developmental biology, Aorta, Endothelium, Vascular/drug effects/metabolism/pathology/ultrasonography, Platelet Glycoprotein GPIb-IX Complex/metabolism, NADPH Oxidase/antagonists & inhibitors/metabolism, Macrophages, Acetophenones, NADPH Oxidases, Acetophenones/pharmacology, Receptors, LDL/genetics/metabolism, Atherosclerosis, Molecular biology, Mice, Inbred C57BL, Oxidative Stress, Disease Models, Animal, chemistry, Receptors, LDL, Antioxidants/pharmacology, Apocynin, Endothelium, Vascular, Atherosclerosis/drug therapy/genetics/metabolism/pathology/ultrasonography, Oxidative stress, Biomarkers

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Contributors: University of Cincinnati (UC), Hospital Infantil de México Federico Gómez (HIMFG), Institut de biologie de Lille - IBL (IBLI), Université de Lille, Sciences et Technologies-Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Université de Lille, Droit et Santé-Centre National de la Recherche Scientifique (CNRS), This work was supported by the National Institutes of Health (grants DK093848 to RJS and R01 DK091425 to YMUL)., Department of Psychiatry and Behavioral Neuroscience [UC, Cincinnati], Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Centre National de la Recherche Scientifique (CNRS)-Université de Lille, Droit et Santé, We thank K.M. Murphy from Washington University and the Howard Hughes Medical Institute for providing the ETV5 KO mouse line., Mécanismes de tumorigenèse et thérapies ciblées, Centre National de la Recherche Scientifique (CNRS)-Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Université de Lille

Source: Physiology and Behavior
Physiology and Behavior, Elsevier, 2015, 150, pp.38-42. ⟨10.1016/j.physbeh.2015.03.027⟩
Physiology and Behavior, Elsevier, 2016, 150, pp.38-42. ⟨10.1016/j.physbeh.2015.03.027⟩

Subject Terms: Blood Glucose, Receptors, Vasopressin, Vasopressin, Corticotropin-Releasing Hormone, MESH: Anti-Inflammatory Agents/pharmacology, [SDV]Life Sciences [q-bio], MESH: Receptors, Vasopressin/metabolism, Anti-Inflammatory Agents, Pituitary-Adrenal System, MESH: Dexamethasone/pharmacology, MESH: Mice, Knockout, Dexamethasone, MESH: Glucocorticoids/blood, Mice, Behavioral Neuroscience, stress, 0302 clinical medicine, Mineralocorticoid receptor, Glucocorticoid receptor, Hypothalamic–pituitary–adrenocortical (HPA) axis, MESH: Receptors, Vasopressin/genetics, Adrenal Glands, Insulin, MESH: Adrenal Glands/pathology, MESH: Animals, MESH: Thymus Gland/pathology, Vasopressin receptor, MESH: Receptors, Glucocorticoid/metabolism, Mice, Knockout, 0303 health sciences, Thymic involution, MESH: Transcription Factors/metabolism, glucocorticoids, [SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism, hypothalamic-pituitary-adrenocortical (HPA) axis, MESH: Hypothalamo-Hypophyseal System/metabolism, MESH: Glucocorticoids/administration & dosage, DNA-Binding Proteins, Hypothalamus, Dexamethasone suppression test, MESH: Receptors, Glucocorticoid/genetics, Glucocorticoid, hormones, hormone substitutes, and hormone antagonists, medicine.drug, MESH: Receptors, Mineralocorticoid/genetics, Hypothalamo-Hypophyseal System, medicine.medical_specialty, 030209 endocrinology & metabolism, Experimental and Cognitive Psychology, MESH: Receptors, Mineralocorticoid/metabolism, Thymus Gland, Biology, MESH: Stress, Psychological/pathology, MESH: Pituitary-Adrenal System/pathology, Article, 03 medical and health sciences, Receptors, Glucocorticoid, Internal medicine, [SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry, Molecular Biology/Genomics [q-bio.GN], medicine, Animals, MESH: Transcription Factors/genetics, MESH: Mice, 030304 developmental biology, MESH: DNA-Binding Proteins/genetics, MESH: DNA-Binding Proteins/metabolism, ETV5, MESH: Gene Expression Regulation/genetics, MESH: Blood Glucose/physiology, Receptors, Mineralocorticoid, Endocrinology, Gene Expression Regulation, MESH: Insulin/blood, MESH: Corticotropin-Releasing Hormone/blood, metabolism, Stress, Psychological, Transcription Factors, MESH: Stress, Psychological/metabolism

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